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BiohacksAI

Evidence-Based Biohacking

Patent Pending

Defective SLCO1B3 causes hyperbilirubinemia, Rotor type (HBLRR)

REACTOME PATHWAY
Reactome: R-HSA-56190581 genes19 compounds

The Defective SLCO1B3 causes hyperbilirubinemia, Rotor type (HBLRR) pathway (Reactome ID: R-HSA-5619058) involves 1 genes and is affected by 19 compounds in the BiohacksAI evidence corpus. Compound-pathway associations are derived from target overlap: a compound is linked to this pathway if it targets ≥2 genes within the pathway.

Genes in this Pathway

SLCO1B3

Compounds Affecting Defective SLCO1B3 causes hyperbilirubinemia, Rotor type (HBLRR)

#CompoundTargets HitStudies
1Bilirubin296
2Arachidonic Acid245
3Genistein300
4Losartan1,000
5Tannins Polyphenolic compounds with molecular weights of around 500-3000 daltons and containing enough hydroxyl groups (1-2 per 100 MW) for effective cross linking of other compounds (ASTRINGENTS).300
6beta Carotene299
7candesartan cilexetil [Supplementary Concept]300
8Mometasone Furoate292
9Olmesartan Medoxomil300
10Ritonavir298
11Atorvastatin1,000
12Antimycin300
13Gemfibrozil297
14Rifamycins300
15Rifamycins300
16Silymarin10
17Taurocholic Acid300
18Triiodothyronine300
19Vincristine299

About the Defective SLCO1B3 causes hyperbilirubinemia, Rotor type (HBLRR) Pathway

The Defective SLCO1B3 causes hyperbilirubinemia, Rotor type (HBLRR) pathway is catalogued in Reactome (ID: R-HSA-5619058) and involves 1 genes. In the BiohacksAI corpus, 19 compounds have documented interactions with at least 2 genes in this pathway, establishing mechanistic relevance. Key pathway genes include SLCO1B3.