BETABiohacksAI is a research tool for informational purposes only. All outputs are computational hypothesis candidates — not confirmed mechanisms, not medical advice, and not a substitute for professional medical judgment. Independent experimental validation is always required.
BiohacksAI is an evolving scientific literature platform. New compounds and evidence are indexed continuously.
B
○
BiohacksAI/Targets/KCNK10/Compounds

Compounds That Target KCNK10

potassium two pore domain channel subfamily K member 10 · 6 compounds in BiohacksAI corpus

KCNK10 (potassium two pore domain channel subfamily K member 10) is a biological target with 6 documented compound interactions in the BiohacksAI corpus. The compounds listed below have been identified through computational analysis of BindingDB binding assay data and PubMed literature. Each interaction is ranked by confidence score reflecting the breadth and strength of experimental evidence. This resource supports research into KCNK10 modulation for therapeutic and biohacking applications.

View KCNK10 target page →UniProt: P57789 ↗
Most Studied Compounds for KCNK10
1
Dha
300+
studies
2
Linoleic Acid
300+
studies
3
Lysophosphatidylcholines
300+
studies
4
Quinidine
298+
studies
5
Carvedilol
296+
studies
6
Arachidonic Acid
245+
studies
All 6 Compounds — Ranked by Evidence
1
Arachidonic Acid
245+
studies
0.69
confidence
2
Dha
300+
studies
0.69
confidence
3
Carvedilol
296+
studies
0.69
confidence
4
Linoleic Acid
300+
studies
0.69
confidence
5
Lysophosphatidylcholines
300+
studies
0.69
confidence
6
Quinidine
298+
studies
0.69
confidence

The compounds above were identified through computational analysis of BindingDB binding assay data and PubMed literature for KCNK10 (potassium two pore domain channel subfamily K member 10). Confidence scores reflect the strength and breadth of experimental evidence.

Explore related resources: KCNK10 target overview · All biological targets · Biological pathways

Data from BindingDB and PubMed. For research purposes only. Not medical advice.